Volume 38, Issue 6, November 2014

Neutralophilic bacteria, especially those affecting human health, possess mechanisms that enhance growth in moderate acid (pH 4–6) and enable survival in extreme acid (pH 1–3).

Beyond being a repair process, SOS induction leads to a very strong but transient response to genotoxic stress, which potentiates bacterial survival and adaptation to changing environments.

We take a closer look at how complement and coagulation induce stress to bacterial cells.

Regulation of protein synthesis under conditions of environmental stress is mediated by a host of ribosome binding factors.

Oral iron administration, host iron status and intestinal inflammation can have a large impact on the gut microbiota composition, metabolism and virulence. This review comprehensively highlights the multifaceted aspects of nutritional iron stress within the colon lumen.

The cellular requirement for metal homeostasis necessitates that successful pathogens elaborate and regulate metal acquisition and detoxification systems in response to host-imposed nutritional immunity.

Bacterial colonizers of human epithelia have a series of means to withstand mechanical removal and other physical stresses, which include in particular surface-attached binding proteins that interact with human matrix components.