Pillars Article: Isolation of a Novel Gene Mutated in Wiskott-Aldrich Syndrome. Cell. 1994. 78: 635–644 Get access

Whole Exome Sequencing (WES) of a Small IPEX-like Patient Cohort results in High Hit Rate and Supports Focusing on the Usual Suspects. Get access

• Abstract

Targeted Gene Editing Restores Regulated CD40L Expression and Function in X-HIGM T cells. Get access

• Abstract

FOXP3 Inhibits Activation-Induced NFAT2 Expression in T Cells Thereby Limiting Effector Cytokine Expression Get access

• Abstract
• Supplementary data

Wiskott-Aldrich Syndrome Protein Is Required for Homeostasis and Function of Invariant NKT Cells Get access

• Abstract

Anti-CD3 antibodies induce tolerance to FVIII in Hemophilia A mice after FVIII plasmid-mediated gene therapy (89.19) Get access

• Abstract

Analysis of FOXP3 Reveals Multiple Domains Required for Its Function as a Transcriptional Repressor Get access

• Abstract

The Wiskott-Aldrich Syndrome Protein Regulates Nuclear Translocation of NFAT2 and NF-κB (RelA) Independently of Its Role in Filamentous Actin Polymerization and Actin Cytoskeletal Rearrangement Free

• Extract

The Wiskott-Aldrich Syndrome Protein Regulates Nuclear Translocation of NFAT2 and NF-κB (RelA) Independently of Its Role in Filamentous Actin Polymerization and Actin Cytoskeletal Rearrangement Get access

• Abstract

Functional Analysis of Peripheral Blood B Cells in Patients with X-Linked Agammaglobulinemia Get access

• Abstract

Purine Nucleoside Phosphorylase Deficiency: A Molecular Model for Selective Loss of T Cell Function Get access

• Abstract

Immunoglobulin Class Responsible for Gonococcal Bactericidal Activity of Normal Human Sera Get access

• Abstract

Antibody Responses to a T Cell Dependent Antigen in C4 Deficiency Get access

• Abstract

Immunologic Reactivity in Canine Marrow Graft Recipients Get access

• Abstract