Graves disease (GD) is a thyroid-specific autoimmune disease and the most common cause of hyperthyroidism. Its pathogenesis is associated with the disruption of immune tolerance and autoantibody production. However, the mechanisms underlying immune abnormalities remain incompletely elucidated. This article investigates changes in the cellular composition and function of peripheral blood mononuclear cells (PBMCs) in GD patients at single-cell resolution.
Latest Published Articles
Historically, 17β-hydroxysteroid dehydrogenase type 3 (HSD17B3) was thought to be the key enzyme responsible for testicular testosterone production. In humans, loss-of-function mutations in HSD17B3 impair testosterone production during prenatal life leading to impaired development of androgen-dependent tissues in 46,XY ...
Context Polycystic ovary syndrome (PCOS) is accompanied by impaired mitochondrial biogenesis in the ovary and uterus. Whether impaired mitochondrial biogenesis exhibits in villi of PCOS, and its effect and underlying mechanism remain unclear. Objective This work aimed to investigate mitochondrial biogenesis status and ...
Endocrine-Disrupting Chemicals (EDCs)
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Why Publish in Endocrinology?
Carol A. Lange, PhD, former Editor-in-Chief of Endocrinology shares why you should submit your research to the flagship basic science journal of the Endocrine Society.
Advice for Getting Published | Carol A Lange, PhD, and Stephen R Hammes, MD, PhD
Endocrinology's former Editor-in-Chief, Carol A Lange, PhD, and Stephen R Hammes, MD, PhD, past Editor-in-Chief of Molecular Endocrinology share critical tips on how to get published.
