
Jim Cassidy (ed.)
et al.
Published online:
01 June 2011
Published in print:
01 May 2010
Online ISBN:
9780191726385
Print ISBN:
9780199563135
Contents
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Genetic factors Genetic factors
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Tumour suppressor genes Tumour suppressor genes
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Proto-oncogenes Proto-oncogenes
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DNA repair genes DNA repair genes
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Specific genes that confer a high probability of susceptibility to specific cancer Specific genes that confer a high probability of susceptibility to specific cancer
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Genes with modest effects that may interact with environmental factors Genes with modest effects that may interact with environmental factors
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Genetic (somatic) mutations caused by recognizable carcinogens causing sporadic cancers Genetic (somatic) mutations caused by recognizable carcinogens causing sporadic cancers
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Gender Gender
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External factors External factors
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Smoking Smoking
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Alcohol Alcohol
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Diet Diet
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Obesity Obesity
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General dietary risk factors General dietary risk factors
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Specific dietary risk factors Specific dietary risk factors
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Exercise Exercise
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Infections Infections
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Viral infections Viral infections
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Human papillomavirus (HPV) Human papillomavirus (HPV)
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Hepatitis B and C virus (HBV, HCV) Hepatitis B and C virus (HBV, HCV)
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Epstein–Barr virus (EBV) Epstein–Barr virus (EBV)
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Bacterial infections Bacterial infections
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Helicobacter pylori Helicobacter pylori
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Parasitic infections Parasitic infections
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Schistosomiasis haematobium (bilharzial bladder disease) Schistosomiasis haematobium (bilharzial bladder disease)
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Exposures Exposures
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Solar exposure Solar exposure
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Other radiation exposure Other radiation exposure
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Other exposures Other exposures
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Industrial exposure Industrial exposure
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Pharmacological exposure Pharmacological exposure
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Environmental exposure Environmental exposure
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Further reading Further reading
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Cite
Cassidy, Jim, and others (eds), 'Aetiology and epidemiology', in Jim Cassidy, and others (eds), Oxford Handbook of Oncology, 3 edn, Oxford Medical Handbooks (Oxford , 2010; online edn, Oxford Academic, 1 June 2011), https://doi.org/10.1093/med/9780199563135.003.02, accessed 9 May 2025.
Extract
Genetic factors
...
Tumour suppressor genes
...
Proto-oncogenes
...
DNA repair genes
...
The relative contribution of the genetic mutation to the cancer varies:
Specific genes that confer a high probability of susceptibility to specific cancer
...
Genes with modest effects that may interact with environmental factors
...
Genetic (somatic) mutations caused by recognizable carcinogens causing sporadic cancers
...
Gender
...
External factors
Smoking
...
Alcohol
...
Diet
Obesity
...
General dietary risk factors
...
Specific dietary risk factors
...
Exercise
...
Infections
Sixteen per cent of the worldwide incidence of cancer is due to infection. For developed countries, the proportion is 9% and for developing countries >20%.
Viral infections
...
Human papillomavirus (HPV)
...
Hepatitis B and C virus (HBV, HCV)
...
Epstein–Barr virus (EBV)
...
Bacterial infections
Helicobacter pylori
...
Parasitic infections
Schistosomiasis haematobium (bilharzial bladder disease)
...
Exposures
Solar exposure
...
Other radiation exposure
...
Other exposures
Subject
Radiation Oncology
Series
Oxford Medical Handbooks
Collection:
Oxford Medicine Online
Disclaimer
Oxford University Press makes no representation, express or implied, that the drug dosages in this book are correct.
Readers must therefore always …
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Oxford University Press makes no representation, express or implied, that the drug dosages in this book are correct.
Readers must therefore always check the product information and clinical procedures with the most up to date published product information and data sheets
provided by the manufacturers and the most recent codes of conduct and safety regulations. The authors and the publishers do not accept responsibility or
legal liability for any errors in the text or for the misuse or misapplication of material in this work. Except where otherwise stated, drug dosages
and recommendations are for the non-pregnant adult who is not breastfeeding.
© Oxford University Press
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