Figure 1
PHACTR1 coronary artery disease risk allele (GG) did not impact on in vivo vascular function. (A) In vivo dilator response to flow (FMD) was not different across the genotypes (GG; P > 0.05, one-way ANOVA, GG = 102, GA = 189, and AA = 138 subjects per genotype). (B) No difference between genotypes was observed in endothelial cell-independent dilation (EID, GG = 73, GA = 142, and AA = 101 subjects per genotype) in response to GTN in vivo. Population was subdivided into non-hypertensive (no HTN). (C) (G = 66, GA = 147, and AA = 105 subjects per genotype) and (D) (G = 61, GA = 134, AA = 94 subjects per genotype) and hypertensive (E) (G = 76, GA = 139, and AA = 97 subjects per genotype) and (F) (G = 33, GA = 83, and AA = 54 subjects per genotype) no difference was observed in either FMD or EID across the genotypes in either population.

PHACTR1 coronary artery disease risk allele (GG) did not impact on in vivo vascular function. (A) In vivo dilator response to flow (FMD) was not different across the genotypes (GG; P > 0.05, one-way ANOVA, GG = 102, GA = 189, and AA = 138 subjects per genotype). (B) No difference between genotypes was observed in endothelial cell-independent dilation (EID, GG = 73, GA = 142, and AA = 101 subjects per genotype) in response to GTN in vivo. Population was subdivided into non-hypertensive (no HTN). (C) (G = 66, GA = 147, and AA = 105 subjects per genotype) and (D) (G = 61, GA = 134, AA = 94 subjects per genotype) and hypertensive (E) (G = 76, GA = 139, and AA = 97 subjects per genotype) and (F) (G = 33, GA = 83, and AA = 54 subjects per genotype) no difference was observed in either FMD or EID across the genotypes in either population.

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