Figure 2
Association between the highest ApoA1 decile and adverse outcomes in sub-groups. Adjusted for age, sex, race, body mass index, hypertension, diabetes, current/former smoking, frequent alcohol use (defined as ≥3 times/week), triglycerides, LDL-C, and eGFR, excluding the variable of stratification for each model. P-values represent the interaction term between variable of stratification and 10th decile of ApoA1 levels (1.91–2.50 g/L), with the 8th decile of ApoA1 levels (1.67–1.75 g/L) being the reference.

Association between the highest ApoA1 decile and adverse outcomes in sub-groups. Adjusted for age, sex, race, body mass index, hypertension, diabetes, current/former smoking, frequent alcohol use (defined as ≥3 times/week), triglycerides, LDL-C, and eGFR, excluding the variable of stratification for each model. P-values represent the interaction term between variable of stratification and 10th decile of ApoA1 levels (1.91–2.50 g/L), with the 8th decile of ApoA1 levels (1.67–1.75 g/L) being the reference.

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