Figure 1
The roles of cytokines in heart failure. Cardiac stressors that initiate and exacerbate cardiac dysfunction evoke leucocyte activation in the heart with cytokine production and release. The associated skeletal muscle deconditioning up-regulates the same cytokines, albeit to a lesser extent. TNF and IL-1β, through IL-6 promote CRP production in the liver. The release of this acute phase reactant appears to be a composite biomarker of the inflammatory cascade activated by heart failure and its levels predict outcome. The inset shows the targeting of cytokines with mixed effects. HF, heart failure; HHF, hospitalization from HF; hsCRP, highly sensitive C-reactive protein.

The roles of cytokines in heart failure. Cardiac stressors that initiate and exacerbate cardiac dysfunction evoke leucocyte activation in the heart with cytokine production and release. The associated skeletal muscle deconditioning up-regulates the same cytokines, albeit to a lesser extent. TNF and IL-1β, through IL-6 promote CRP production in the liver. The release of this acute phase reactant appears to be a composite biomarker of the inflammatory cascade activated by heart failure and its levels predict outcome. The inset shows the targeting of cytokines with mixed effects. HF, heart failure; HHF, hospitalization from HF; hsCRP, highly sensitive C-reactive protein.

Close
This Feature Is Available To Subscribers Only

Sign In or Create an Account

Close

This PDF is available to Subscribers Only

View Article Abstract & Purchase Options

For full access to this pdf, sign in to an existing account, or purchase an annual subscription.

Close