Table 1

Study characteristics

Study, yearPatients (n)Age ± stdMale %PopulationaReference standardTechnique usedbTime between imaging modalities
Weinsaft 20161120156 ± 1284.1ADE (delayed enhancement)-CMR1 + 224 h
Weinsaft 2009312161.2 ± 13.376.9BDE-CMR1 + 2Within 7 days
Weinsaft 20111824360 ± 1563.4CDE-CMR1Within 7 days
Delewi 20121919456.1 ± 9.484.9Acine-CMR124 h
Garg 201220481N/RN/RBCE (contrast enhanced)-CMR1 + 2Within 7 days
Meurin 2015217859.1 ± 12.172ADE-CMR124 h
Sürder 20152211356.8 ± 10.288.3ADE-CMR124 h
Chaosuwannakit 20212320660.2 ± 14.268.4CDE-CMR1
Kim 2017247454 ± 1191.2ADE-CMR324 h
Phan 201925210N/R85ADE-CMR1Within 7 days
Kim 201426192N/RN/RADE-CMR1 + 224 h
Study, yearPatients (n)Age ± stdMale %PopulationaReference standardTechnique usedbTime between imaging modalities
Weinsaft 20161120156 ± 1284.1ADE (delayed enhancement)-CMR1 + 224 h
Weinsaft 2009312161.2 ± 13.376.9BDE-CMR1 + 2Within 7 days
Weinsaft 20111824360 ± 1563.4CDE-CMR1Within 7 days
Delewi 20121919456.1 ± 9.484.9Acine-CMR124 h
Garg 201220481N/RN/RBCE (contrast enhanced)-CMR1 + 2Within 7 days
Meurin 2015217859.1 ± 12.172ADE-CMR124 h
Sürder 20152211356.8 ± 10.288.3ADE-CMR124 h
Chaosuwannakit 20212320660.2 ± 14.268.4CDE-CMR1
Kim 2017247454 ± 1191.2ADE-CMR324 h
Phan 201925210N/R85ADE-CMR1Within 7 days
Kim 201426192N/RN/RADE-CMR1 + 224 h

aA = post-MI, B = mixed (e.g. post-MI, heart failure, stroke). C = left ventricular systolic dysfunction.

b1 = non-contrast TTE, 2 = contrast TTE, 3 = apical wall motion scoring. Data that was not reported (N/R) in the primary paper was assumed to be missing at random.

Table 1

Study characteristics

Study, yearPatients (n)Age ± stdMale %PopulationaReference standardTechnique usedbTime between imaging modalities
Weinsaft 20161120156 ± 1284.1ADE (delayed enhancement)-CMR1 + 224 h
Weinsaft 2009312161.2 ± 13.376.9BDE-CMR1 + 2Within 7 days
Weinsaft 20111824360 ± 1563.4CDE-CMR1Within 7 days
Delewi 20121919456.1 ± 9.484.9Acine-CMR124 h
Garg 201220481N/RN/RBCE (contrast enhanced)-CMR1 + 2Within 7 days
Meurin 2015217859.1 ± 12.172ADE-CMR124 h
Sürder 20152211356.8 ± 10.288.3ADE-CMR124 h
Chaosuwannakit 20212320660.2 ± 14.268.4CDE-CMR1
Kim 2017247454 ± 1191.2ADE-CMR324 h
Phan 201925210N/R85ADE-CMR1Within 7 days
Kim 201426192N/RN/RADE-CMR1 + 224 h
Study, yearPatients (n)Age ± stdMale %PopulationaReference standardTechnique usedbTime between imaging modalities
Weinsaft 20161120156 ± 1284.1ADE (delayed enhancement)-CMR1 + 224 h
Weinsaft 2009312161.2 ± 13.376.9BDE-CMR1 + 2Within 7 days
Weinsaft 20111824360 ± 1563.4CDE-CMR1Within 7 days
Delewi 20121919456.1 ± 9.484.9Acine-CMR124 h
Garg 201220481N/RN/RBCE (contrast enhanced)-CMR1 + 2Within 7 days
Meurin 2015217859.1 ± 12.172ADE-CMR124 h
Sürder 20152211356.8 ± 10.288.3ADE-CMR124 h
Chaosuwannakit 20212320660.2 ± 14.268.4CDE-CMR1
Kim 2017247454 ± 1191.2ADE-CMR324 h
Phan 201925210N/R85ADE-CMR1Within 7 days
Kim 201426192N/RN/RADE-CMR1 + 224 h

aA = post-MI, B = mixed (e.g. post-MI, heart failure, stroke). C = left ventricular systolic dysfunction.

b1 = non-contrast TTE, 2 = contrast TTE, 3 = apical wall motion scoring. Data that was not reported (N/R) in the primary paper was assumed to be missing at random.

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